Cannabiskonsum im Jugend- und jungen Erwachsenenalter geht mit häufigeren psychoseähnlichen Erfahrungen einher. Diese Erfahrungen ähneln möglicherweise den Symptomen einer Psychose, erfüllen jedoch normalerweise nicht die klinischen Schwellenwerte.

I’ve linked to the news release in the post above. In this comment, for those interested, here’s the link to the peer reviewed journal article:

https://www.cambridge.org/core/journals/psychological-medicine/article/relationship-between-recreational-cannabis-use-psychoticlike-experiences-and-the-salience-network-in-adolescent-and-young-adult-twins/D4A158FEF185AF8923CB3535A7EDF58E

From the linked article:

New research published in Psychological Medicine suggests that recreational **cannabis use during adolescence and young adulthood is associated with more frequent psychotic-like experiences. These experiences may resemble symptoms of psychosis but do not typically meet clinical thresholds**. However, the study also finds that this association is not explained by structural changes in a key brain network involved in salience processing. The findings point toward unique environmental influences on both cannabis use and the emergence of these unusual thoughts or perceptions.

The results suggested that cannabis use was linked to more frequent psychotic-like experiences. This association remained even after controlling for age, sex, and intelligence. Among those who had used cannabis, both the total number of psychotic-like experiences and the number of positive experiences (such as delusional thoughts or hallucinations) were higher compared to non-users.

One of the six brain connectivity factors—the one related to the anterior cingulate cortex and insula—was also associated with psychotic-like experiences. These brain areas are known to be involved in assigning importance to internal and external stimuli, which is thought to play a role in psychotic symptoms.

However, when the researchers tested whether this brain factor explained the link between cannabis use and psychotic-like experiences, they found no evidence for a mediation effect. In other words, while cannabis use and this brain connectivity pattern were each separately associated with psychotic-like experiences, the connectivity pattern did not appear to be the pathway through which cannabis use exerted its influence.

The twin analysis provided additional insight. It showed that individual environmental factors, rather than genetics or shared family environment, were the main influences on both cannabis use and psychotic-like experiences. This means that even though these traits were associated, they likely developed independently within each person, shaped by their unique experiences.

They talk about individual environmental factors, what does that mean?

Since we’re not talking about shared environments or genetics, are they saying that people who are individually predisposed toward one are also more predisposed toward the other?

This message was paid for by BIG PHARMA.. Total BS.