Neue Forschungsergebnisse zeigen, dass, nachdem die Abwehrkräfte des Körpers das Virus hinter COVID-19 abgetötet haben, übrig gebliebene verdaute Brocken des SARS-CoV-2-Spike-Proteins aufgrund ihrer Form bestimmte Immunzellen angreifen können. „Zombie“-Coronavirus-Fragmente können die Aktivität von Molekülen des körpereigenen Immunsystems imitieren, um Entzündungen voranzutreiben.
https://newsroom.ucla.edu/releases/covid-19-viral-fragments-target-kill-specific-immune-cells
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COVID-19 viral fragments shown to target and kill specific immune cells in UCLA-led study
Clues about extreme cases and omicron’s effects come from a cross-disciplinary international research team
Key takeaways
A UCLA-led research team demonstrated that when human immune enzymes break up the spike protein of the virus behind COVID-19, some resulting fragments have the ability to punch holes in membranes of human immune cells.
Those fragments target and kill specific cells based on their shape — the same types of sentinel cells and killer cells depleted in severe COVID-19.
Fragments of protein from the omicron variant showed less activity against the immune cells, a finding that may account for why it’s less dangerous than other strains.
**New research shows that after the body’s defenses kill the virus behind COVID-19, leftover digested chunks of SARS-CoV-2 spike protein can target specific immune cells based on their shape**. The revelations could explain why certain populations of cells that detect and fight infection are depleted in patients with severe COVID-19, and shed light on the omicron variant’s milder symptoms.
The study, published in the Proceedings of the National Academy of Sciences, may launch a line of inquiry that informs new strategies for quelling the most serious symptoms of COVID-19. Led by a UCLA team, the scientific collaboration comprises nearly three dozen engineers, microbiologists, immunologists, chemists, physicists, medical researchers and analytical experts. Authors are based at universities, medical centers and national laboratories and institutes in the United States, China, Germany, India and Italy. The research was funded in part by the National Science Foundation and the National Institutes of Health.
The team’s findings build on an earlier UCLA discovery identifying “**zombie” coronavirus fragments that can imitate the activity of molecules from the body’s own immune system to drive inflammation**. Now, not only have the researchers shown that human immune enzymes can break down the SARS-CoV-2 spike protein into such fragments, they found that some fragments can work together to attack important types of immune cells by targeting their cell shapes.
For those interested, here’s the link to the peer reviewed journal article:
https://www.pnas.org/doi/10.1073/pnas.2521841122
I have no clue what that means in detail, but it reads very bad.
If someone has a text for the scientific illiterate normies, like me, I’ll be very happy.
From an evolutionary standpoint, have we ever seen a virus with as many features as the Sars-cov-2 ?
A month after I recovered from COVID I was diagnosed out of nowhere with mono in my mid 30s which resulted in severe liver inflammation, bells palsy, chronically swollen lymph nodes and up to this point an unending 15 month (NDPH) headache. Prior to COVID I had 0 health issues. Based on the results of this study would it be possible that COVID compromised my immune system to make me more susceptible to both getting Mono and the wrath of chronic health conditions I’ve dealt with since?
This is the second study I’ve seen indicating the spike protein alone can be dangerous, but my understanding is the mRNA COVID vaccine makes your cells produce spike protein which the immune system then learns to destroy.
But nobody mentions when they talk about spike protein being dangerous, how or if this might apply to the risk profile of vaccines that contain or have your body produce this potentially dangerous protein?